Latest Amyloid precursor protein Stories
Much of the debate in Alzheimer's disease (AD) has focused on whether the protein amyloid-beta or the tau protein is the symptom or the cause of the disease.
A study in mice shows how a breakdown of the brain's blood vessels may amplify or cause problems associated with Alzheimer's disease.
Patients with traumatic brain injury (TBI) had increased deposits of β-Amyloid (Aβ) plaques, a hallmark of Alzheimer Disease (AD), in some areas of their brains in a study by Young T. Hong, Ph.D., of the University of Cambridge, England, and colleagues.
With the flick of a light switch, researchers at the Salk Institute for Biological Studies can change the shape of a protein in the brain of a mouse, turning on the protein at the precise moment they want.
Two rare mutations in the human gene that lead to the most common, late-onset variant of Alzheimer's have been identified by Chair Member of the Cure Alzheimer's Disease Research Consortium,
A leading group of Alzheimer's researchers contends that, as biomarkers to detect signals of the disease improve at providing clinically meaningful information, researchers will need guidance on how to constructively disclose test results and track how disclosure impacts both patients and the data collected in research studies.
In patients with early Alzheimer's disease, disruptions in brain networks emerge about the same time as chemical markers of the disease appear in the spinal fluid.
An element that is essential to all living organisms could also be one of the primary environmental factors that can lead to the onset of Alzheimer’s disease, according to new research appearing in Monday’s edition of the journal Proceedings of the National Academy of Sciences.
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