Latest Amyloid precursor protein Stories
Scientists at the University of Liverpool and Callaghan Innovation in New Zealand have developed a new chemical approach to help harness the natural ability of complex sugars to treat Alzheimer's disease.
Many Alzheimer’s disease researchers are focused on stopping the buildup of amyloid plaques, dense protein deposits that are a telltale sign of the debilitating disease. However, a group of scientists from the Salk Institute for Biological Studies decision to think outside the box and pursue a different treatment strategy has paid off.
New research published Thursday in the journal Neuron sheds new light on the molecular causes of Alzheimer’s disease, while also revealing a potential new therapy which could help prevent cognitive decline and brain damage during the early stages of the neurodegenerative disorder.
Evidence indicates that the accumulation of amyloid-beta proteins, which form the plaques found in the brains of Alzheimer's patients, is critical for the development of Alzheimer's disease, which impacts 5.4 million Americans.
Researchers reporting in Tuesday’s issue of JAMA have identified a new gene mutation that nearly doubles the risk of developing Alzheimer’s disease in older blacks. The findings come from the largest genome-wide, government-funded study on Alzheimer’s genes in the African-American community.
A new genetically engineered lab rat that has the full array of brain changes associated with Alzheimer's disease supports the idea that increases in a molecule called beta-amyloid in the brain causes the disease.
The Journal of Neuroscience has published a study led by researchers at the Max Planck Florida Institute for Neuroscience, the first and only U.S. extension of the prestigious Max Planck Society, that may hold a stunning breakthrough in the fight to treat Alzheimer's disease.
"Use it or lose it." The saying could apply especially to the brain when it comes to protecting against Alzheimer's disease.
New findings by Columbia researchers suggest that along with amyloid deposits, white matter hyperintensities (WMHs) may be a second necessary factor for the development of Alzheimer's disease.
- Emitting flashes of light; glittering.