Latest Biochemistry of Alzheimer's disease Stories
Researchers may be one step closer to slowing the onset and progression of Alzheimer's disease.
New imaging tool could eventually lead to earlier detection among pre-symptomatic individuals.
Scientists have determined that a new instrument known as PIB-PET is effective in detecting deposits of amyloid-beta protein plaques in the brains of living people, and that these deposits are predictive of who will develop Alzheimerâ€™s disease.
Nearly 20 years ago Huntington Potter kicked up a storm of controversy with the idea that Down syndrome and Alzheimerâ€™s were the same disease -- Now the evidence is in: He was right.
TGen-led team finds 3 proteins that dismantle 'bridges' within brain cells.
Olfactory dysfunction in mouse model may correlate to humans.
Current Alzheimer's disease (AD) research indicates that accumulation of amyloid-beta (AÎ²) protein plaques in the brain is central to the development of AD.
Aging is the single greatest risk factor for Alzheimer's disease. In their latest study, researchers at the Salk Institute for Biological Studies found that simply slowing the aging process in mice prone to develop Alzheimer's disease prevented their brains from turning into a neuronal wasteland.
Alzheimer's disease is caused by the build-up of a brain peptide called amyloid-beta. That's why eliminating the protein has been the focus of almost all drug research pursuing a cure for the devastating neurodegenerative condition.
Protein reduces levels of amyloid beta and tau hyperphosphorylation, 2 hallmarks of Alzheimer's.
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