Latest Blood lipids Stories
International research teams uncover multiple genetic risk factors and demonstrate how a non-coding gene variant can contribute to heart disease.
A global team of researchers co-led by the University of Michigan School of Public Health has discovered or confirmed 95 regions of the human genome where genetic variants are associated with blood cholesterol and triglyceride levels, which are major indicators of heart disease risk.
Together with their colleagues from the Global Lipids Genetics Consortium, scientists of Helmholtz Zentrum MÃ¼nchen have found 95 gene loci in the human genome, each of which is associated with at least one of the four most important factors of lipoprotein metabolism: total cholesterol, LDL cholesterol, HDL cholesterol and triglycerides.
Researchers have uncovered 95 genes that are believed to affect blood cholesterol, including a few that are affected by drugs and others that might be the basis of new drugs.
Most people know that high cholesterol levels place them at increased risk for heart disease and stroke.
Thousands of children with high levels of LDL cholesterol are going untreated, as current guidelines exclude as many as 10-percent of those who could benefit from medication.
A US study conducted in mice suggests the amount of cholesterol that circulates through the bloodstream is partially regulated by the brain, which counters previous beliefs that it is solely controlled by what we eat and by cholesterol production in the liver.
Cholesterol crystals lead to life-threatening inflammation in blood vessel walls.
Consuming a higher amount of added sugars in processed or prepared foods is associated with lower levels of high-density lipoprotein cholesterol (HDL-C, the "good cholesterol") and higher levels of triglycerides, which are important risk factors for cardiovascular disease.
Mice that are missing a protein involved in the response to low oxygen stay lean and healthy, even on a high-fat diet, a new study has found.
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