Latest Creutzfeldt–Jakob disease Stories
Researchers said on Wednesday that villagers in the highlands of Papua New Guinea who ritualistically ate human brains but did not die of a brain disease called kuru have a genetic mutation that protects them.
The regulating protein Srebp2 drives cholesterol formation, which prions need for their propagation, in prion-infected neuronal cells.
Specific cells within the immune system could help explain why younger people are more susceptible to variant CJD, scientists believe.
A collaboration between scientists at Vanderbilt University and the University of California, San Francisco has led to the first direct information about the molecular structure of prions.
A University of Toronto-led team of scientists says it has determined the evolutionary origin of the prion gene. The researchers said the diseased prion proteins are responsible for the fatal neurodegenerative Creutzfeldt-Jakob disease in humans, bovine spongiform encephalopathy, also known as mad cow
A University of Toronto-led team has uncovered the evolutionary ancestry of the prion gene, which may reveal new understandings of how the prion protein causes diseases such as bovine spongiform encephalopathy (BSE), also known as "mad cow disease."
According to a study published on Wednesday, it appears that a variety of prion disease found in deer may be spread through the fecal matter of infected animals even when they exhibit no symptoms of the illness.
A new study shows that nervous system integrity and axonal properties may play a key role in prion diseases.
A British expert on the human form of mad cow disease says it is essential that coroners test the brains and spleens of all young people who die. Professor John Collinge says without post-mortem tests for the infection it is impossible to get accurate information on how many people in Britain may be carrying it, The Times of London reported Wednesday.
Data from an ongoing multi-year study suggest that people who consume deer and elk with chronic wasting disease (CWD) may be protected from infection by an inability of the CWD infectious agent to spread to people.
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