Latest Daniel Geschwind Stories
UCLA researchers have combined two tools – gene expression and the use of peripheral blood -- to expand scientists' arsenal of methods for pinpointing genes that play a role in autism.
Mice with a single defective gene are showing striking parallels to humans with autism. According to a study published in the September 30th issue of Cell, a Cell Press publication, the mouse model offers several promising discoveries.
Mice with a defective version of a single gene show behaviors and symptoms that are remarkably similar to characteristics observed in humans with autism spectrum disorders.
No cure exists for frontotemporal dementia, which strikes between the ages of 40 and 64 and accounts for at least one in four cases of early-onset dementia.
For decades, autism researchers have faced a baffling riddle: how to unravel a disorder that leaves no known physical trace as it develops in the brain.
Many gene variants have been linked to autism, but how do these subtle changes alter the brain, and ultimately, behavior?
If humans are genetically related to chimps, why did our brains develop the innate ability for language and speech while theirs did not?
Six million years ago, chimpanzees and humans diverged from a common ancestor and evolved into unique species. Now UCLA scientists have identified a new way to pinpoint the genes that separate us from our closest living relative and make us uniquely human.
- Growing in low tufty patches.