Quantcast
Last updated on April 23, 2014 at 11:18 EDT

Latest Dimitri Krainc Stories

2013-07-31 12:25:41

Northwestern's neurology and neurosurgery program climbs to 7th in the nation and remains highest ranked in Illinois and Chicago CHICAGO, July 31, 2013 /PRNewswire-USNewswire/ -- Northwestern Memorial Hospital's neurology and neurosurgery program has been ranked as 7th in the country on the U.S. News & World Report 2013-14 Best Hospitals specialty rankings released on Tuesday, July 16. This is the first year Northwestern Memorial has been recognized in the top 10 for neurology...

2011-12-23 06:48:49

(Ivanhoe Newswire) — Treatment that increases brain levels of a crucial regulatory enzyme may slow the loss of brain cells that characterize Huntington´s disease (HD) and other neurodegenerative disorders, according to this study. Increased expression of Sirt1, one of a family of enzymes called sirtuins, in the brain of a mouse model of HD protected against neurodegeneration. They also identified a potential mechanism for this protective effect. "Diseases such as Huntington's,...

2011-12-19 11:43:13

Mutant huntingtin protein appears to block activity of Sirt1, suggesting potential treatment target Treatment that increases brain levels of an important regulatory enzyme may slow the loss of brain cells that characterizes Huntington's disease (HD) and other neurodegenerative disorders. In a report receiving advance online publication in Nature Medicine, a Massachusetts General Hospital (MGH)-based research team reports that increased expression of Sirt1, one of a family of enzymes called...

2009-04-02 13:03:43

A new study has identified a potential strategy for removing the abnormal protein that causes Huntington's disease (HD) from brain cells, which could slow the progression of the devastating neurological disorder. In the April 3 issue of Cell, a team of researchers from the MassGeneral Institute for Neurodegenerative Disease (MGH-MIND) describes how an alteration to the mutated form of the huntingtin protein appears to accelerate its breakdown and removal through normal cellular...