Latest DNA damage theory of aging Stories
There are many reasons our bodies age. The process is determined by an accumulation of various kinds of cell damage that impair the function of bodily organs.
University of Texas Medical Branch at Galveston researchers have found a surprising connection between a key DNA-repair process and a cellular signaling network linked to aging, heart disease, cancer and other chronic conditions.
A game-changing find challenges previously held beliefs about the role of mutations in cancer development. Researchers at the Fred Hutchinson Cancer Research Center in Seattle say their findings show that the number of new mutations is significantly lower in cancers than in normal cells.
James D. Watson, Frances Crick, and Maurice Wilkins were awarded the Nobel Prize in Physiology or Medicine in 1962 for their discovery of the structure of DNA.
Cells in the human body are constantly being exposed to stress from environmental chemicals or errors in routine cellular processes.
A study that tracked genetic mutations through the human equivalent of about 5,000 years has demonstrated for the first time that oxidative DNA damage is a primary cause of the process of mutation - the fuel for evolution but also a leading cause of aging, cancer and other diseases.
Deleting two genes in mice responsible for repairing DNA strands damaged by oxidation leads to several types of tumors, providing additional evidence that such stress contributes to the development of cancer.
Scientists at St. Jude Children's Research Hospital have teased apart the biological details distinguishing two related neurological diseasesâ€”ataxia telangiectasia-like disease (ATLD) and Nijmegen breakage syndrome (NBS).
St. Jude scientists' findings in ataxia telangiectasia-like disease and Nijmegen breakage syndrome offer insight into the links between brain disease and cancer vulnerability in people carrying the diseases MEMPHIS, Tenn., Jan. 14 /PRNewswire-USNewswire/ -- Scientists at St.
When Dr. Susan Rosenberg, professor of molecular and human genetics at Baylor College of Medicine, first published her finding that the mutation rate increased in bacteria stressed by starvation, sometimes resulting in a rare change that benefited the bacteria, it was controversial.
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