Latest DNA repair Stories
Identification of two new proteins in the Fanconi anemia DNA repair pathway may help explain genetic instability in people with Fanconi anemia and how otherwise healthy people are susceptible to cancer from environmentally triggered DNA damage.
A quest that began over a decade ago with a chance observation has reached a milestone: the identification of a gene that may regulate regeneration in mammals.
Program Will Include Paid Internship Program for Students WASHINGTON, March 8 /PRNewswire-USNewswire/ -- The Justice Department today announced a new internship program that will employ students part-time to conduct intensive outreach efforts with Native Americans and their families whose work in the uranium industry during the Cold War benefited the United States but exposed them to radiation and may entitle them to compensation under the Radiation Exposure Compensation Act (RECA).
Adding a new link to our understanding of the complex chain of chemistry that keeps living cells alive
DNA damage sensor also responds to oxidative harm outside the nucleus
PLEASANTON, Calif., Feb. 17 /PRNewswire/ -- Roche Molecular Systems, Inc. (SIX: RO, ROG; OTCQX: RHHBY) announced today a research collaboration with Merck Sharp & Dohme Corp.
A discovery by Newcastle University experts could provide the next step in fighting age related diseases, such as diabetes and heart disease.
Scientists have shown that cells' DNA-reading machinery can skim through certain kinds of damaged DNA without skipping any letters in the genetic "text."
The research group of Dr. FranÃ§ois Robert, a researcher at the Institut de recherches cliniques de MontrÃ©al (IRCM), in collaboration with the team of Dr. Daniel Durocher (Samuel Lunenfeld Research Institute and University of Toronto) accomplished a technical breakthrough: they mapped all the fragile sites of a living organism, the budding yeast Saccharomyces cerevisiae.
A team of researchers, led by Pier Paolo Pandolfi, at Beth Israel Deaconess Medical Center, Boston, has identified a new type of cellular senescence (i.e., irreversible arrest of cell growth) and determined a way to enhance it to suppress prostate tumor development and growth in mice.