Latest DNA repair Stories
Mutations in a gene called XRCC2 cause increased breast cancer risk.
The well-being of living cells requires specialized squads of proteins that maintain order.
A protein abundantly found in treatment-resistant cancers holds an important tumor-suppressor out of the cell nucleus, where it would normally detect DNA damage and force defective cells to kill themselves, a team of scientists reports in the current Cancer Cell.
A new prospective study of patients with Lynch syndrome – an inherited disorder of cancer susceptibility caused by mutations in specific DNA repair genes – provides the first strong evidence that people with Lynch syndrome face significantly increased risks of breast and pancreatic cancers.
It's been more than 50 years since James Watson and Francis Crick showed that DNA is a double helix of two strands that complement each other.
Scientists have demonstrated for the first time that certain cells circulating in human blood – so-called monocytes – are extremely sensitive to reactive oxygen species (ROS).
An inherited mutation in a gene known as the guardian of the genome is likely the link between exploding chromosomes and some particularly aggressive types of cancer, scientists at the European Molecular Biology Laboratory (EMBL), the German Cancer Research Centre (DKFZ) and the University Hospital, all in Heidelberg, Germany, have discovered.
The chemotherapy drugs required to push a common form of adult leukemia into remission may contribute to DNA damage that can lead to a relapse of the disease in some patients, findings of a new study suggest.
Scientists have found at least one instance when the smaller sibling gets to call the shots and cancer patients may one day benefit.
Aging-related tissue degeneration can be caused by mitochondrial dysfunction in tissue stem cells.
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