Latest Huntington's disease clinical research Stories
Novel ZFP Therapeutic Approach Selectively Represses Expression of Huntington's Disease-Causing Gene Variant in Patient-Derived Cells and Animal Models RICHMOND, Calif., Nov.
Researchers believe they have learned how mutations in the gene that causes Huntington’s disease kill brain cells, a finding that could open new opportunities for treating the fatal disorder.
A new study published 26th November in the open access journal PLOS Biology, identifies a new target in the search for therapeutic interventions for Huntington's disease – a devastating late-onset neurodegenerative disorder.
Recycling is not only good for the environment, it's good for the brain. A study using rat cells indicates that quickly clearing out defective proteins in the brain may prevent loss of brain cells.
Alzheimer's, Huntington's, Parkinson's. Names forever linked to what they represent: diseases that ravage the brain's neurons and leave entire regions to wither and die.
A new light-based technique for measuring levels of the toxic protein that causes Huntington's disease (HD) has been used to demonstrate that the protein builds up gradually in blood cells.
Researchers recently announced that they were able to produce stem cells from skin cells from a person who had severe, early-onset form of Huntington Disease.
Using a human induced pluripotent stem cell (iPSC) from a patient suffering from Huntington’s disease, researchers at the Buck Institute for Research on Aging have corrected the genetic mutation that is responsible for the disease.
More than 15,000 Americans have Huntington’s disease, a fatal condition marked by uncontrolled movements and cognitive and psychiatric problems. Currently, there are no available treatments to alter the effects of Huntington’s disease, but a new study brings researchers one step closer to finding one.
A new gene-silencing strategy can reverse core symptoms associated with Huntington's disease.
- Growing in low tufty patches.