Latest Microglia Stories
Two proteins conspire to promote a lethal neurological disease.
A small amount of exercise shields older animals from memory loss following a bacterial infection.
Gardeners know that some trees require regular pruning: some of their branches have to be cut so that others can grow stronger.
NEW YORK, June 22, 2011 /PRNewswire-USNewswire/ -- The Alzheimer's Drug Discovery Foundation (ADDF) announced today that it has awarded a grant to Axxam SpA to develop small molecules to treat Alzheimer's disease by targeting inflammation. (Logo: http://photos.prnewswire.com/prnh/20090805/DC57332LOGO) The award will fund a drug discovery project to identify compounds that block the puringeric receptor, P2X7, which is involved in inflammation in the brain.
Vision scientists have identified a key player in macular degeneration (MD), raising hope for a treatment for the currently incurable blinding disease.
Neurodegenerative diseases like Alzheimer's and Parkinson's are partly attributable to brain inflammation.
Transplanting stem cells derived from umbilical cord blood cells and menstrual blood cells may offer future therapeutic benefit for those suffering from stroke, Alzheimer's disease, and amyotrophic lateral sclerosis (ALS).
Targeting the neuroinflammatory causes of Parkinson's disease with a naturally present brain chemical signal could offer a better understanding of the clinical mechanisms of the disease and open a future therapeutic window.
A new target for the prevention of adverse immune responses identified as factors in the development of Alzheimer's disease (AD) has been discovered by researchers at the University of South Florida's Department of Psychiatry and the Center of Excellence for Aging and Brain Repair.
Immune cells known as microglia, long thought to be activated in the brain only when fighting infection or injury, are constantly active and likely play a central role in one of the most basic, central phenomena in the brain â€“ the creation and elimination of synapses.
- A hairdresser.