Latest Motor neuron Stories
Stretch sensors in our muscles participate in reflexes that serve the subconscious control of posture and movement.
WASHINGTON, June 29, 2014 /PRNewswire-USNewswire/ -- In work supported by The ALS Association, researchers have shown that increasing the clearance of misfolded protein from neurons improves
The entire process of eating is a basic biological task. The tongue and jaw perform a well-choreographed ballet where the tongue positions your food for the teeth and recedes to its ready position just in time for the jaw to close down onto it.
Imagine you cannot move your eyes up, and you cannot lift your upper eyelid. You walk through life with your head tilted upward so that your eyes look straight when they are rolled down in the eye socket.
A new way to artificially control muscles using light, with the potential to restore function to muscles paralyzed by conditions such as motor neuron disease and spinal cord injury, has been developed by scientists at UCL and King’s College London.
By studying nerve cells that originated in patients with a severe neurological disease, a University of Wisconsin-Madison researcher has pinpointed an error in protein formation that could be the root of amyotrophic lateral sclerosis.
Keck School of Medicine of USC neuroscientists have unlocked a piece of the puzzle in the fight against Lou Gehrig's disease, a debilitating neurological disorder that robs people of their motor skills.
In most cases of amyotrophic lateral sclerosis (ALS), or Lou Gehrig's disease, a toxin released by cells that normally nurture neurons in the brain and spinal cord can trigger loss of the nerve cells affected in the disease.
We might have more in common with a lamprey than we think.
Normally muscles contract in order to support the body, but in a rare condition known as cataplexy the body's muscles "fall asleep" and become involuntarily paralyzed.