Latest Myofibroblast Stories
Patients with damaged organs could be helped by new treatments after scientists have discovered how tissues scar.
A team of scientists that includes Saint Louis University researchers has identified a new way to intervene in the molecular and cellular cascade that causes fibrosis – a condition where the body's natural process of forming scars for wound healing goes into overdrive and causes diseases.
An international team of scientists, led by researchers at the University of California, San Diego School of Medicine, report that significant numbers of myofibroblasts – cells that produce the fibrous scarring in chronic liver injury – revert to an inactive phenotype as the liver heals.
A synthesised compound which is also found in bear bile could help prevent disturbances in the heart's normal rhythm.
Idiopathic pulmonary fibrosis (IPF) affects about 100,000 people in the US each year and is fatal within three years of diagnosis.
An invasive cell that leads to fibrosis of the lungs may be stopped by cutting off its supply of sugar.
Scientists have identified a protein that plays a key role in debilitating changes that occur in the heart after a heart attack.
In the latest of a series of related papers, researchers at the University of California, San Diego School of Medicine, with colleagues in Austria and elsewhere, present a new and more definitive explanation of how fibrotic cells form, multiply and eventually destroy the human liver, resulting in cirrhosis.
In-growth and new generation of blood vessels, which must take place if a wound is to heal or a tumor is to grow, have been thought to occur through a branching and further growth of a vessel against a chemical gradient of growth factors.