Latest Neprilysin Stories
- Preclinical data demonstrates reduction of Alzheimer's brain deficits to lower toxic Amyloid-beta and restored synaptic loss - PLANTATION, Fla., May 28, 2014 /PRNewswire/ -- Neurotrope,
UC Irvine neurobiologists have found that genetically modified neural stem cells show positive results when transplanted into the brains of mice with the symptoms and pathology of Alzheimer's disease.
Small genetic mutations that add up over time could create an evolutionary express lane that leads to the rapid development of new traits.
New molecular tools developed at the University of Michigan show promise for "cleansing" the brain of amyloid plaques, implicated in Alzheimer's disease.
Reducing a protein called beta-amyloid in young mice with a condition resembling Down syndrome improves their ability to learn, researchers at UT Southwestern Medical Center have found.
Saint Louis University researchers find low doses of protein linked to Alzheimer's disease enhances memory in healthy.
U.S. scientists say the protein neprilysin can reduce plaque formation and neuron death associated with Alzheimer's disease, but at the expense of lifespan. Thomas Jefferson University researchers said the buildup of amyloid-beta protein plaques within the brain is a hallmark of Alzheimer's.
U.S. medical scientists say they have identified a gene that puts people at risk for developing Alzheimer's disease.
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