Latest Neurofibrillary tangle Stories
The Capital Institute for Neurosciences, part of Capital Health, Hopewell Township, New Jersey, launches new program to treat Alzheimer's disease and other cognitive disorders.
Results could lead to improved surgery care and a better understanding of the long-term effects of anesthesia and surgery on a senior adult's post-operative thinking. Boston
Building on research published eight years ago in the journal Chemistry and Biology, Kenneth S. Kosik, Harriman Professor in Neuroscience and co-director of the Neuroscience Research Institute (NRI) at UC Santa Barbara, and his team have now applied their findings to two distinct, well-known mouse models, demonstrating a new potential target in the fight against Alzheimer's and other neurodegenerative diseases.
A new genetically engineered lab rat that has the full array of brain changes associated with Alzheimer's disease supports the idea that increases in a molecule called beta-amyloid in the brain causes the disease.
Goldilocks was on to something when she preferred everything "just right."
Researchers from Boston University School of Medicine (BUSM) have identified a novel group of proteins that accumulate in the brains of patients with Alzheimer's disease.
According to a new study, the neuron-killing pathology of Alzheimer's disease (AD), which begins before clinical symptoms appear, requires the presence of both amyloid-beta (a-beta) plaque deposits and elevated levels of an altered protein called p-tau.
Immunotherapy with the antibody bapineuzumab in patients with mild to moderate Alzheimer disease resulted in decreases in a cerebrospinal fluid biomarker, which may indicate downstream effects on the degenerative process.
Repeated stress triggers the production and accumulation of insoluble tau protein aggregates inside the brain cells of mice.
- An armed gangster.