Latest Protective autoimmunity Stories
Results presented in poster presentation beginning today and in oral presentation Saturday, November 8, at 1:40 PM at Society for Immunotherapy of Cancer annual meeting. ANN ARBOR,
DIEPENBEEK, Belgium, September 4, 2014 /PRNewswire/ -- Scientists discover how to 'switch off' autoimmune diseases Apitope, the drug discovery
The cause of neuronal death in Parkinson's disease is still unknown, but a new study proposes that neurons may be mistaken for foreign invaders and killed by the person's own immune system, similar to the way autoimmune diseases like type I diabetes, celiac disease, and multiple sclerosis attack the body's cells.
Apoptosis is used by cells that are changed by disease or are simply not needed any longer to eliminate themselves before they become a hazard to the body—on a cellular level, death is part of life.
Led by Dr. Peiyee Lee and Dr. Richard Gatti, researchers at the Eli and Edythe Broad Center of Regenerative Medicine and Stem Cell Research at UCLA have used induced pluripotent stem (iPS) cells to advance disease-in-a-dish modeling of a rare genetic disorder, ataxia telangiectasia (A-T).
Neurons that control hunger in the central nervous system also regulate immune cell functions, implicating eating behavior as a defense against infections and autoimmune disease development.
Over the previous several years, medical professionals have noted and documented marked increases in the prevalence of autoimmune diseases. An international team of scientists and researchers have come together in an attempt to identify the cause behind the increase in such diseases.
Researchers from Queen Mary, University of London have discovered that two proteins which are believed to play a key role in controlling the body's immune response are found in lower levels in T lymphocytes from patients with multiple sclerosis (MS).
In many pathologies of the nervous system, there is a common event - cells called microglia are activated from surveillant watchmen into fighters.
- A gift; a largess; a gratuity; a present; a dole.