Latest Reactive oxygen species Stories
An international team of scientists, led by Monash University researchers, has found that anti-oxidants commonly touted for their health-promoting benefits, could contribute to the early onset of Type 2 diabetes.
We've all heard about the damage that reactive oxygen species (ROS) â€“ aka free radicals â€“ can do to our bodies and the sales pitches for antioxidant vitamins, skin creams or "superfoods" that can stop them. In fact, there is considerable scientific evidence that chronic ROS production within cells can contribute to human diseases, including insulin resistance and type 2 diabetes.
For years, health conscious people have been taking antioxidants to reduce the levels of reactive oxygen in their blood and prevent the DNA damage done by free radicals, which are the result of oxidative stress. But could excessive use of antioxidants deplete our immune systems?
Research published in the journal Genetics suggests new ways to stop byproducts from the air we breathe from harming our muscles.
Scientists at the Burnham Institute for Medical Research in California say they've discovered reactive oxygen plays a key role in cancer metastasis. The researchers, led by Professor Sara Courtneidge, said they determined reactive species, such as superoxide and hydrogen peroxide, help form invadopodia -- cellular protrusions implicated in cancer cell migration.
The growth of cancerous tumors is fueled, at least in part, by the buildup of free radicalsâ€”highly reactive oxygen-containing molecules.
With all the hype about beneficial antioxidants in everything from face cream to cereal bars, you'd think their targetsâ€”oxygen radicalsâ€”must be up to no good. It's true, the buildup of oxygen radicals and other reactive oxygen species (ROS) in cells contributes to aging and possibly to diseases such as cancer and Alzheimer's.
Scientists in Michigan are reporting the development of a powerful new probe for identifying proteins affected by a key chemical process important in aging and disease.
A potent antioxidant enzyme made in the mitochondria reduces or prevents the memory and learning impairments of Alzheimerâ€™s disease in animals and could provide new direction to treating the disease, said researchers from Baylor College of Medicine and New York University in a new report.
Mice with a defective mitochondrial protein called MCLK1 produce elevated amounts of reactive oxygen when young; that should spell disaster, yet according to a study in this week's JBC these mice actually age at a slower rate and live longer than normal mice.
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