Latest Rett syndrome Stories
When cells divide normally, DNA gets copied perfectly and distributed among the daughter cells with an even hand.
A paper published online today in Nature reveals that glia play a key role in preventing the progression of the most prominent Rett Syndrome symptoms displayed by mouse models of the disease: lethality, irregular breathing and apneas, hypoactivity and decreased dendritic complexity.
NEW YORK, June 15, 2011 /PRNewswire/ -- Huda Y.
The protein MeCP2 is porridge to the finicky neuron.
An IRSF funded study published today in the journal Science has shown that the childhood disorder Rett syndrome, can be reestablished in adult animals by "switching off" a critical disease causing gene in healthy adult animals.
Model of Rett syndrome suggests a deficit in processing cues from the environment.
Since 1999, scientists have known that a mutation in a gene called MeCP2 (methyl-CpG-Binding protein 2) can cause Rett syndrome, a neurodevelopmental disorder.
Study marks the emergence of disease-modifying treatments for autism spectrum disorders BOSTON, Dec.
With few exceptions, jumping genes-restless bits of DNA that can move freely about the genome-are forced to stay put.
A groundbreaking study published Friday in the leading scientific journal, Cell, revealed that a team of investigators had successfully generated nerve cells using skin cells from four individuals with Rett syndrome.
- The analysis of literature, focusing on the words and grammar to the exclusion of context or literary merit.