Latest Skeletal muscle Stories
Cells rely on tiny molecular motors to deliver cargo, such as mRNA and organelles, within the cell. The critical nature of this transport system is evidenced by the fact that disruption of motors by genetic defects leads to fatal diseases in humans.
A research collaboration between Munich-based biophysicists and a structural biologist in Hamburg is helping to explain why our muscles, and those of other animals, don't simply fall apart under stress.
Scientists in the UK and Denmark have shown that if elderly men were given growth hormone and exercised their legs showed an appreciable muscle mass increase.
Exercise requires the integrated activity of every organ and tissue in the body, and understanding how these respond to the decreased oxygen levels present at moderate to high altitude is the focus of the current special issue of High Altitude Medicine & Biology, a peer-reviewed journal published by Mary Ann Liebert, Inc.
New research shows that muscle atrophy is a much more ordered and deliberate process than previously thought.
Researchers have found a potential new treatment for the common problem of muscle atrophy. Results of the animal study were presented at The Endocrine Society's 91st Annual Meeting in Washington, D.C.
Two signals â€“ an external one from retinoic acid and an internal one from the transcription factor Neurogenin2 â€“ cooperate to activate chromatin (the basic material of chromosomes) and help determine that certain nerve progenitor cells become motor neurons, said researchers from Baylor College of Medicine (www.bcm.edu) in a report in the current issue of the journal Neuron.
During desperate times, such as fasting, or muscle wasting that afflicts cancer or AIDS patients, the body cannibalizes itself, atrophying and breaking down skeletal muscle proteins to liberate amino acids.
Scientists at the Ottawa Hospital Research Institute (OHRI) and the University of Ottawa have discovered a powerful new way to stimulate muscle regeneration, paving the way for new treatments for debilitating conditions such as muscular dystrophy.
New study shows heart muscle protein can replace its missing skeletal muscle counterpart to give mice with myopathy a long and active life.