Latest Tau protein Stories
Amyloid-beta and tau protein deposits in the brain are characteristic features of Alzheimer disease.
Increasing puromycin-sensitive aminopeptidase, the most abundant brain peptidase in mammals, slowed the damaging accumulation of tau proteins that are toxic to nerve cells and eventually lead to the neurofibrillary tangles, a major pathological hallmark of Alzheimer's disease and other forms of dementia.
A new study is unraveling the earliest events associated with neurodegenerative diseases characterized by abnormal accumulation of tau protein.
Dynamic regulation of the chaperone protein Hsp27 was required to get rid of abnormally accumulating tau in the brains of mice genetically modified to develop the memory-choking tau tangles associated with Alzheimer's disease, a University of South Florida-led study found.
Scientists from Berlin, Bonn and Dundee show in animal models that the diabetes drug Metformin has an effect against one of the main causes of the Alzheimerâ€™s disease.
Researchers from Georgetown University Medical Center's departments of neuroscience, pharmacology, physiology, biochemistry, molecular and cellular biology, and the interdisciplinary program of neuroscience will present more than 50 research abstracts at the 40th Annual Meeting of the Society for Neuroscience Nov. 13th through the 17th in San Diego.
NEW YORK, Nov. 11, 2010 /PRNewswire/ -- Oligomerix, Inc.
Finding a drug that can cross the blood-brain barrier is the bane of drug development for Alzheimerâ€™s disease and other neurological disorders of the brain.
Cells that help to protect the central nervous system may also contribute to pathological changes in the brain.
A new study uncovers a protein modification that may contribute to the formation of neuron-damaging neurofibrillary tangles in the human brain.
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