Latest Telomerase Stories
BLUE BELL, Pa., June 17, 2011 /PRNewswire/ -- Inovio Pharmaceuticals, Inc.
Researchers have found new clues about aging, by identifying a new pathway that sets the clock for programmed aging in normal cells.
National Institutes of Health researchers have identified a new pathway that sets the clock for programmed aging in normal cells.
NEW YORK, June 8, 2011 /PRNewswire/ -- When expressed (turned on), the gene (hTERT) located on Chromosome number 5p15.33 activates the enzyme telomerase. Human cells can keep living and dividing indefinitely when telomerase is continually present; i.e. the cells become immortal.
A rare genetic disease called dyskeratosis congenita, caused by the rapid shortening of telomeres (protective caps on the ends of chromosomes), can be mimicked through the study of undifferentiated induced pluripotent stem cells.
Cancer researchers at UT Southwestern Medical Center are helping unlock the cellular-level function of the telomerase enzyme, which is linked to the disease's growth.
Adults with post-traumatic stress disorder and a history of childhood trauma had significantly shorter telomere length than those with PTSD but without childhood trauma.
Scientists have uncovered an important clue to one of the mechanisms underlying cancer cell immortality.
A study of DNA rearrangements in roundworm chromosomes may offer new insight into large-scale genome duplications that occur in developing tumors.
Animals that reproduce asexually by somatic cloning have special mechanisms that delay ageing provide exceptionally good health.
- The analysis of literature, focusing on the words and grammar to the exclusion of context or literary merit.