Latest Telomere Stories
National Institutes of Health researchers have identified a new pathway that sets the clock for programmed aging in normal cells.
Cutting edge research and experts are agreeing that slowing aging is a real possibility. But how to do that is not always clear with all the variety of information out there.
NEW YORK, June 8, 2011 /PRNewswire/ -- When expressed (turned on), the gene (hTERT) located on Chromosome number 5p15.33 activates the enzyme telomerase. Human cells can keep living and dividing indefinitely when telomerase is continually present; i.e. the cells become immortal.
Research by influential doctors and biologists such as Aubrey de Grey, PhD are increasingly showing that longer, more vital lives are possible in effect, turning the average middle age to around 100. One of the key factors that is found in all the research is telomere length.
A rare genetic disease called dyskeratosis congenita, caused by the rapid shortening of telomeres (protective caps on the ends of chromosomes), can be mimicked through the study of undifferentiated induced pluripotent stem cells.
Study of institutionalized Romanian children finds prematurely shortened telomeres, a mark of cell aging BOSTON, May 17, 2011 /PRNewswire-USNewswire/ -- Studies in institutionalized Romanian children have found that the length of time spent in conditions of social deprivation and neglect correlates with lower IQ and behavioral problems.
A new blood test is going on sale this year that will predict how long someone has to live, based on their speed of aging.
Cancer researchers at UT Southwestern Medical Center are helping unlock the cellular-level function of the telomerase enzyme, which is linked to the disease's growth.
Adults with post-traumatic stress disorder and a history of childhood trauma had significantly shorter telomere length than those with PTSD but without childhood trauma.
Scientists have uncovered an important clue to one of the mechanisms underlying cancer cell immortality.