Latest Telomere Stories
Scientists at Johns Hopkins have provided more clues to one of the least understood phenomena in some cancers: why the "ends caps" of cellular DNA, called telomeres, lengthen instead of shorten.
Two Baylor College of Medicine doctors have received support for their research from Alex's Lemonade Stand, a nonprofit group dedicated to fighting pediatric cancer.
BLUE BELL, Pa., June 17, 2011 /PRNewswire/ -- Inovio Pharmaceuticals, Inc.
Researchers have found new clues about aging, by identifying a new pathway that sets the clock for programmed aging in normal cells.
National Institutes of Health researchers have identified a new pathway that sets the clock for programmed aging in normal cells.
Cutting edge research and experts are agreeing that slowing aging is a real possibility. But how to do that is not always clear with all the variety of information out there.
NEW YORK, June 8, 2011 /PRNewswire/ -- When expressed (turned on), the gene (hTERT) located on Chromosome number 5p15.33 activates the enzyme telomerase. Human cells can keep living and dividing indefinitely when telomerase is continually present; i.e. the cells become immortal.
Research by influential doctors and biologists such as Aubrey de Grey, PhD are increasingly showing that longer, more vital lives are possible in effect, turning the average middle age to around 100. One of the key factors that is found in all the research is telomere length.
A rare genetic disease called dyskeratosis congenita, caused by the rapid shortening of telomeres (protective caps on the ends of chromosomes), can be mimicked through the study of undifferentiated induced pluripotent stem cells.
Study of institutionalized Romanian children finds prematurely shortened telomeres, a mark of cell aging BOSTON, May 17, 2011 /PRNewswire-USNewswire/ -- Studies in institutionalized Romanian children have found that the length of time spent in conditions of social deprivation and neglect correlates with lower IQ and behavioral problems.