Latest Tumor suppressor genes Stories
Angelina Jolie’s recent news of opting to have a double mastectomy because she has the BRCA1 gene mutation shows how pertinent organizations such as Bright Pink® are to educating women
Australian Researchers Astonished to Learn P53 Not Only Seeks and Destroys, But Prevents DALLAS (PRWEB) May 19, 2013 Scientists are on the verge of unraveling
Jean LaMantia, Registered Dietitian, Cancer Survivor and Author of The Essential Cancer Treatment Nutrition Guide and Cookbook, advises that
A particular tumor suppressor gene that fights cancer cells does more than clamp down on unabated cell division -- the hallmark of the disease -- it also can help make cells more fit by allowing
Researchers at the Stanford University School of Medicine have identified a group of proteins that are mutated in about one-fifth of all human cancers.
Cancer cells are a problem for the body because they multiply recklessly, refuse to die and blithely metastasize to set up shop in places where they don't belong.
Researchers at Moffitt Cancer Center have found that a deficiency in an important anti-tumor protein, p53, can slow or delay DNA repair after radiation treatment.
Researchers of the hereditary cancer research group at the Bellvitge Biomedical Research Institute (IDIBELL) and the Catalan Institute of Oncology (ICO) conducted a functional and structural study of seven missense variants of the BRCA1 gene concluding that three of these variants are pathogenic, linked to the risk of suffering breast or ovarian cancer.
The US Supreme Court will hear oral arguments on Monday in a case that could determine whether or not human DNA can be patented, which could have a tremendous impact on genetic research, one way or the other.
Mutations in a protein called SPOP (speckle-type POZ protein) disarm it, allowing another protein called steroid receptor coactivator-3 (SRC-3) to encourage the proliferation and spread of prostate cancer cells.
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