Latest Virus latency Stories
While new treatments have proven successful at suppressing HIV infection, they have thus far been unable to eliminate it due to the fact that they cannot attack the virus as it hides dormant in the cells of a person’s immune system.
polyDNA answers new questions on herpes, HSV, herpes virus, genital sores, etc.
A team of researchers at the University of North Carolina at Chapel Hill have successfully flushed latent HIV infection from hiding, with a drug used to treat certain types of lymphoma.
In what may prove to be a major step forward for the treatment of HIV-1 infection, scientists have discovered an effective way to eliminate a notoriously persistent form of the virus that does not respond to current therapies.
--Children's Hospital of Philadelphia Study Sheds Light on How HIV Takes Over Cell Cycle -- PHILADELPHIA, Feb.
New research identifies a molecular mechanism that the human immunodeficiency virus (HIV) appears to utilize for generating random fluctuations called "noise" in its gene expression.
Random fluctuations in gene expression can influence the fates of cells infected with human immunodeficiency virus (HIV) far more than previously thought, according to new research from Howard Hughes Medical Institute (HHMI) researchers at the University of California, Berkeley. By combining experimental and computational studies of HIV's replication cycle, the researchers found evidence that the virus may become latent in some cells by harnessing the random molecular behavior of the cell.
Herpes simplex virus 1 and 2 (HSV-1 and HSV-2), are two members of the virus family Herpesviridae that infect humans. They are both ubiquitous and contagious and can spread when an infected person is producing and shedding the virus. Symptoms include watery blisters in the skin, mouth, lips, or genitals. Lesions usually heal with a scab characteristic of herpetic disease. HSV-1 and -2 remain latent in the body hiding from the immune system in the cell bodies of nerves. Some people do...